Medical Knowledge Base

Atomic Knowledge Catalog

Study single-topic medical articles (etiology, pathogenesis, diagnosis, treatment) across specialties and ICD-linked conditions.

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Showing 24 of 120

EtiologyK04.5

Chronic Apical Periodontitis — Etiology

Silent periradicular destruction — granuloma and cyst formation at the root apex

dentistrychronic-apical-periodontitis
Open article
EtiologyK05.3

Chronic Periodontitis — Etiology

Progressive irreversible destruction of the tooth-supporting apparatus

dentistrychronic-periodontitis
Open article
EtiologyK04.0

Chronic Pulpitis — Etiology

Prolonged, low-grade pulp inflammation progressing silently toward irreversibility

dentistrychronic-pulpitis
Open article
EtiologyK04.7

Dental Abscess — Etiology

Acute purulent infection with soft tissue spread — from localized pus to life-threatening fascial space infection

dentistrydental-abscess
Open article
EtiologyK02.9

Dental Caries — Etiology

The most prevalent infectious disease worldwide

dentistrydental-caries
Open article
EtiologyK03.2

Dental Erosion — Etiology

Irreversible loss of dental hard tissue by chemical dissolution from non-bacterial acids

dentistrydental-erosion
Open article
EtiologyK00.3

Dental Fluorosis — Etiology

Enamel hypomineralization from excessive fluoride ingestion during tooth development

dentistrydental-fluorosis
Open article
EtiologyK03.89

Dentinal Hypersensitivity — Etiology

Sharp, transient pain from exposed dentin in response to thermal, evaporative, tactile, or osmotic stimuli

dentistrydentinal-hypersensitivity
Open article
EtiologyK05.0

Gingivitis — Etiology

Reversible gingival inflammation — the most prevalent oral disease and precursor to periodontitis

dentistrygingivitis
Open article
EtiologyK05.22

Pericoronitis — Etiology

Inflammation around the crown of a partially erupted tooth

dentistrypericoronitis
Open article
EtiologyK05.21

Periodontal Abscess — Etiology

Acute purulent infection localised within the periodontal tissues

dentistryperiodontal-abscess
Open article
EtiologyK04.8

Radicular Cyst — Etiology

The most common odontogenic cyst, arising at the apex of a non-vital tooth

dentistryradicular-cyst
Open article
PathogenesisK04.4

Acute Apical Periodontitis — Pathogenesis

Acute inflammation of the periradicular tissues — from hyperemia to suppuration at the root apex

dentistryacute-apical-periodontitis
Open article
PathogenesisK04.0

Acute Pulpitis — Pathogenesis

Reversible and irreversible inflammatory conditions of the dental pulp

dentistryacute-pulpitis
Open article
PathogenesisK12.0

Aphthous Stomatitis — Pathogenesis

Recurrent painful oral ulcers of unknown etiology — the most common oral mucosal disease

dentistryaphthous-stomatitis
Open article
PathogenesisK04.5

Chronic Apical Periodontitis — Pathogenesis

Silent periradicular destruction — granuloma and cyst formation at the root apex

dentistrychronic-apical-periodontitis
Open article
PathogenesisK05.3

Chronic Periodontitis — Pathogenesis

Progressive irreversible destruction of the tooth-supporting apparatus

dentistrychronic-periodontitis
Open article
PathogenesisK04.0

Chronic Pulpitis — Pathogenesis

Prolonged, low-grade pulp inflammation progressing silently toward irreversibility

dentistrychronic-pulpitis
Open article
PathogenesisK04.7

Dental Abscess — Pathogenesis

Acute purulent infection with soft tissue spread — from localized pus to life-threatening fascial space infection

dentistrydental-abscess
Open article
PathogenesisK02.9

Dental Caries — Pathogenesis

The most prevalent infectious disease worldwide

dentistrydental-caries
Open article
PathogenesisK03.2

Dental Erosion — Pathogenesis

Irreversible loss of dental hard tissue by chemical dissolution from non-bacterial acids

dentistrydental-erosion
Open article
PathogenesisK00.3

Dental Fluorosis — Pathogenesis

Enamel hypomineralization from excessive fluoride ingestion during tooth development

dentistrydental-fluorosis
Open article
PathogenesisK03.89

Dentinal Hypersensitivity — Pathogenesis

Sharp, transient pain from exposed dentin in response to thermal, evaporative, tactile, or osmotic stimuli

dentistrydentinal-hypersensitivity
Open article
PathogenesisK05.0

Gingivitis — Pathogenesis

Reversible gingival inflammation — the most prevalent oral disease and precursor to periodontitis

dentistrygingivitis
Open article